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Our results indicate that DDA complexes with GSH-AuNCs and readily induces their complete transfer to chloroform or dichloromethane (typical solvents to prepare PLGA nanoparticles) as well as to other hydrophobic solvents (hexane and toluene) as shown in Fig
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477–479 This complex interplay between VEGF signaling, endothelial function, and blood pressure highlights challenges in managing anti-VEGF therapy-induced hypertension, warranting further research for effective strategies
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