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Membrane binding alters the properties of MBP, promoting self-interaction into a densely packed protein phase that occupies the major dense lines and tightly binds the cytoplasmic surface of the bilayer together to limit the entry of most proteins into the myelin sheaths ( Raasakka et al., 2017

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Thus, as a general understanding, in the context of experimental therapeutics, increasing model complexity—characterized by the engagement of multiple molecular targets and biological pathways—has to be associated with a proportional increase in the risk of adverse effects [1]