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Therapeutic effects at late regeneration phase (24–72 h in mice) also need to be experimentally verified
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Moreover, SIRT1 H363Y, a catalytically dead mutant of which histidine (H) at site 363 was replaced with tyrosine (Y) 44 , failed to decrease STX17 acetylation, whereas its interaction with STX17 was comparable with that of WT SIRT1 (Fig
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