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Deficiency in Se was shown to inhibit enzyme activity and mRNA expression levels of cytosolic GPx, thereby increasing lipid peroxidation in bovine arterial endothelial cells (BAEC) [71]
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This occurred also as the restored neuromuscular junction function antagonized the effect of the neuromuscular blocker succinylcholine, thereby opposing the inability of the muscle cell to repolarize, and opposing the desensitization at the nerve terminal [102]
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