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These effects were largely attributable to the BH4-dependent endothelial nitric oxide synthase (eNOS) pathways (eNOS dysfunction induces glucose intolerance and insulin resistance) [9,26]
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[DOI] [PMC free article] [PubMed] [Google Scholar] 7.Treitinger A., Spada C., Masokawa I.Y., Verdi J.C., Van Der Sander Silveira M., Luis M.C., Reis M., Ferreira S.I., Abdalla D.S
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